In January 2005 it was reported that cancer has surpassed heart disease - for the first time - as the top killer of Americans aged under 85. In 2002, the most recent year for which information is available, 476009 Americans aged under 85 died of cancer, compared with 450637 died of heart disease. It is estimated that 1372910 new cases of cancer and 570260 cancer deaths are expected this year.
Paclitaxel, a preferred treatment for breast cancer and lung cancer, a risk of cancer promotion and….
Lung cancer remains the biggest cancer killer, projected to claim 163510 lives this year. Paclitaxel will be used in the attempt to save the lives of many of these patients. However, a little-known effect of Paclitaxel is that in a subset of these patients, there will be up to a fivefold increase in the production of interleukin - 8 (IL-8) - a cellular communication molecule that triggers growth of new blood vessels to feed the growth of cancer. In other words, if you fall into this subset of patients, treatment using paclitaxel alone may not be effective in preventing recidivism.
NF-kB blockade enhances the ability to kill cancer Paclitaxel!
IL-8 is under the control of an inflammatory regulating protein called nuclear factor - kappa Beta (NF-kB). During the activation of NF-kB is blocked, IL-8 sec, a bit like a faucet that has been deactivated. Thus, blocking NF-kB activation enhances the ability to kill cancer Paclitaxel. These results were seen with many types of cancer cells, including lung cancer and esophageal cancer cells.
Paclitaxel is not only drug that promotes excessive NF-kB
Paclitaxel is just one of a group of drugs which has the unwanted side effect of the activation of NF-kB. Other drugs in this group include doxorubicin, 5-fluorouracil, cisplatin, VP-16 (etoposide), ARA-C, and Methotrexate. In addition, research shows that excessive NF-kB activity contributes to cancer development in the types of cancers: non-small cell lung cancer, pancreas, liver primary cancer of the head and neck, prostate, breast, esophagus, stomach, colon, Hodgkin's disease and multiple myeloma.
A treatment of chemotherapy, which improves the efficiency… ..
Paclitaxel, along with the other NF-kB activation of chemotherapy drugs, was approved for the treatment of a wide range of cancers. It seems likely that they will continue to be used for the foreseeable future. If you are (or plan to use) Paclitaxel or one another in this group of drugs for the treatment of cancer, there is a symptomatic treatment that you need to know about that improves the effectiveness of these drugs and reduces your risk of a recurrence of cancer.
We have a multi-dimensional approach to reduction of inflammation and Complements Improves the impact of these drugs!
At the learning centre healing in Ann Arbor, MI, where I practice integrative medicine and behavioral oncology, I focus on multi-dimensional ways to empower patients to evaluate and change patterns of eating, behaving , reflection and adaptation which are expected to contribute For the inflammatory reactions in the body. These methods complement the effects of the assassination of cancer Paclitaxel, doxorubicin, 5-FU, and other drugs.
Can clarify genomic tests your specific inflammatory molecular mechanisms that support your cancer
Inflammation is present before, and during the life of a cancer. In cancer, inflammation is a pathological process characterized by injury or destruction of tissues caused by a series of chemical reactions and cellular. It is usually translates into typical signs of pain, heat, redness, swelling and loss of function. However, inflammation is also essential for tissue repair and reconstruction of tissue. Genomic Tests (easily performed with samples of saliva or blood) allows us to create a custom map of your inflammatory tendencies based on your genomic predispositions. This method is revolutionary because it allows you to regulate your genomic capabilities to your advantage, which allows you to reduce the expression of your genes related to inflammation.
What cancer patients need to do is Reducing the expression of genes related to inflammation
Once you know your plan for specific genomic excessive inflammation, we are working together to develop the tools you need to re-define the term of your plan inflammation. These tools must be unique to you, precisely because your abilities expression of genomes are yours. These tools include anti-inflammatory regimes supported by oral and intravenous nutrients that block and down-regulating NF-kB. Remember, this protein is responsible for the abnormal elevation of IL-8 during Paclitaxel administration. By measuring markers of inflammation cell before, during and after chemotherapy treatment, and using your unique tools, we compile a personalized treatment of inflammatory responses record (normal and abnormal), which serves as a reference for the risk of cancer recurrence after treatment of chemotherapy.
With these guidelines personally, you insider knowledge about food choices, attitudes and interpersonal relationships that will be conducive to keeping your genes related to inflammation silent. Patients with high inflammatory markers during chemotherapy are at higher risk for recurrence, and should be monitored more closely and modulate their expression NF-kB after the chemotherapy.
What is important to understand is that:
As your doctor-coach, I recommend that you become the master of your team healing, and that me and my team partner with you in order to clarify the specific molecular mechanisms behind the wheel of your cancer. We coach with the skills and master the tools needed to reduce the collective contribution of food, their feelings and attitudes to the excessive expression of genes associated with inflammation. By working together, we learn how to adjust your pattern inflammation by modulating the expressive ability of NF-kB. The modulation of the expression of your NF-kB is the game of self-discovery, consciousness expansion, forgiveness, and cells (self-) renewal is what allows healing to occur. (To see diagrams describing NF-kB in health and disease, visit our website at http://www.arond-thomasonline.com!)
Learn about how you can improve your chances of not having a cancer recurrence…
You or a loved one can learn about how you can improve the efficiency of your cancer treatments and improve your chances of not having a recurrence. At http://www.1CancerCoach.com.
Paclitaxel, a preferred treatment for breast cancer and lung cancer, a risk of cancer promotion and….
Lung cancer remains the biggest cancer killer, projected to claim 163510 lives this year. Paclitaxel will be used in the attempt to save the lives of many of these patients. However, a little-known effect of Paclitaxel is that in a subset of these patients, there will be up to a fivefold increase in the production of interleukin - 8 (IL-8) - a cellular communication molecule that triggers growth of new blood vessels to feed the growth of cancer. In other words, if you fall into this subset of patients, treatment using paclitaxel alone may not be effective in preventing recidivism.
NF-kB blockade enhances the ability to kill cancer Paclitaxel!
IL-8 is under the control of an inflammatory regulating protein called nuclear factor - kappa Beta (NF-kB). During the activation of NF-kB is blocked, IL-8 sec, a bit like a faucet that has been deactivated. Thus, blocking NF-kB activation enhances the ability to kill cancer Paclitaxel. These results were seen with many types of cancer cells, including lung cancer and esophageal cancer cells.
Paclitaxel is not only drug that promotes excessive NF-kB
Paclitaxel is just one of a group of drugs which has the unwanted side effect of the activation of NF-kB. Other drugs in this group include doxorubicin, 5-fluorouracil, cisplatin, VP-16 (etoposide), ARA-C, and Methotrexate. In addition, research shows that excessive NF-kB activity contributes to cancer development in the types of cancers: non-small cell lung cancer, pancreas, liver primary cancer of the head and neck, prostate, breast, esophagus, stomach, colon, Hodgkin's disease and multiple myeloma.
A treatment of chemotherapy, which improves the efficiency… ..
Paclitaxel, along with the other NF-kB activation of chemotherapy drugs, was approved for the treatment of a wide range of cancers. It seems likely that they will continue to be used for the foreseeable future. If you are (or plan to use) Paclitaxel or one another in this group of drugs for the treatment of cancer, there is a symptomatic treatment that you need to know about that improves the effectiveness of these drugs and reduces your risk of a recurrence of cancer.
We have a multi-dimensional approach to reduction of inflammation and Complements Improves the impact of these drugs!
At the learning centre healing in Ann Arbor, MI, where I practice integrative medicine and behavioral oncology, I focus on multi-dimensional ways to empower patients to evaluate and change patterns of eating, behaving , reflection and adaptation which are expected to contribute For the inflammatory reactions in the body. These methods complement the effects of the assassination of cancer Paclitaxel, doxorubicin, 5-FU, and other drugs.
Can clarify genomic tests your specific inflammatory molecular mechanisms that support your cancer
Inflammation is present before, and during the life of a cancer. In cancer, inflammation is a pathological process characterized by injury or destruction of tissues caused by a series of chemical reactions and cellular. It is usually translates into typical signs of pain, heat, redness, swelling and loss of function. However, inflammation is also essential for tissue repair and reconstruction of tissue. Genomic Tests (easily performed with samples of saliva or blood) allows us to create a custom map of your inflammatory tendencies based on your genomic predispositions. This method is revolutionary because it allows you to regulate your genomic capabilities to your advantage, which allows you to reduce the expression of your genes related to inflammation.
What cancer patients need to do is Reducing the expression of genes related to inflammation
Once you know your plan for specific genomic excessive inflammation, we are working together to develop the tools you need to re-define the term of your plan inflammation. These tools must be unique to you, precisely because your abilities expression of genomes are yours. These tools include anti-inflammatory regimes supported by oral and intravenous nutrients that block and down-regulating NF-kB. Remember, this protein is responsible for the abnormal elevation of IL-8 during Paclitaxel administration. By measuring markers of inflammation cell before, during and after chemotherapy treatment, and using your unique tools, we compile a personalized treatment of inflammatory responses record (normal and abnormal), which serves as a reference for the risk of cancer recurrence after treatment of chemotherapy.
With these guidelines personally, you insider knowledge about food choices, attitudes and interpersonal relationships that will be conducive to keeping your genes related to inflammation silent. Patients with high inflammatory markers during chemotherapy are at higher risk for recurrence, and should be monitored more closely and modulate their expression NF-kB after the chemotherapy.
What is important to understand is that:
- There is an optimal amount of the expression of NF-kB compatible with health;
- Expression excessive contributes to the emergence of diseases like cancer, especially when NF-kB is switched on continuously, and
- You have the power and tools to regulate NF-kB expression.
As your doctor-coach, I recommend that you become the master of your team healing, and that me and my team partner with you in order to clarify the specific molecular mechanisms behind the wheel of your cancer. We coach with the skills and master the tools needed to reduce the collective contribution of food, their feelings and attitudes to the excessive expression of genes associated with inflammation. By working together, we learn how to adjust your pattern inflammation by modulating the expressive ability of NF-kB. The modulation of the expression of your NF-kB is the game of self-discovery, consciousness expansion, forgiveness, and cells (self-) renewal is what allows healing to occur. (To see diagrams describing NF-kB in health and disease, visit our website at http://www.arond-thomasonline.com!)
Learn about how you can improve your chances of not having a cancer recurrence…
You or a loved one can learn about how you can improve the efficiency of your cancer treatments and improve your chances of not having a recurrence. At http://www.1CancerCoach.com.
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